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(2) Children aged 3 2)3- 10/0 had a history of minor trauma or upper respiratory tract infection before onset. Sudden onset, the child refers to pain in the knee joint and thigh, which turns to pain in the hip joint a few days later, and then causes limping, hypothermia, fever and tenderness in the hip joint. The affected hip joint maintains slight flexion and adduction, and the pronation and abduction activities of the hip joint are obviously limited. Compared with the photos of the hip joint, the epiphysis and cartilage surface are not smooth.
(3) patients with dermatoses (such as psoriasis and erythema multiforme). Treatment with steroid drugs can slow down the synthesis of osteoblasts, hinder the transformation of pre-osteoblasts into osteoblasts, and absorb shadow 9 into calcium from the intestine, leading to osteoporosis. After trauma, fractures may occur, resulting in decreased resistance and bone compression or collapse. Due to the compression of bone marrow cells and capillaries, the obstruction of blood supply will lead to osteonecrosis. If it occurs in the femoral head, it is femoral head necrosis. When you find that you are limping when you walk, relieved after a rest, and feel uncomfortable or sore when you sit down, you should probably be alert to this disease.
What sequela does femoral head necrosis have? Subchondral fracture of femoral head, separation of trabecular bone from cartilage, multiple fracture-like bright band changes of femoral head, and normal or basically normal appearance of femoral head. After treatment, 95% patients were completely cured and took part in work like normal people. Only 5% patients did not rest well, walked too much, and collapsed by 0.5- 1mm, resulting in the sequelae of short-term mild claudication. But after half a year or 1 year of treatment, the sequelae of this limping will gradually disappear.
Sequelae of femoral head necrosis include subchondral fracture of femoral head, separation of trabecular bone from cartilage, multiple fracture-like transparent banded changes of femoral head, multiple deceptive or banded sclerotic high-density bone changes of femoral head, collapse of femoral head, flat hypertrophy, reduction or disappearance of trabecular bone, vertical collapse height ≤5mm, and joint gap of about 2 mm. ..
The sequelae of the third stage necrosis of the femoral head are fracture of the femoral head, disappearance or mostly disappearance of trabecular bone, many large-area cystic light transmission changes in the femoral head, many sclerosing high-density bone changes, serious collapse deformation of the femoral head, flat hypertrophy, vertical collapse height >: 5mm, and joint space narrower than 2mm or disappearance.