Secondly, after patients with liver cancer have fever symptoms, corresponding treatment measures should be taken according to the specific causes of liver cancer fever, such as:
(1) If the fever is caused by bacterial infection, it should be treated with anti-inflammatory drugs in time. Hyperthermia should be controlled in time (drug cooling or physical cooling can be used);
(2) If the patient has fever after interventional therapy, and the body temperature does not exceed 38.5℃, and the patient can tolerate it, it is not necessary to cool down temporarily to facilitate the absorption of necrotic tissue. But pay attention to the patient's warmth and ventilation in the ward. In addition, we should closely observe the temperature changes and carry out physical cooling (mix 60% medical alcohol and 40% warm water, wipe the chest, back, soles of feet, palms or use cold ice packs, etc.). ) When symptoms of high fever appear, it should be given immediately.
In addition, patients with liver cancer should pay attention to keep warm and strengthen other nursing work, such as ensuring good indoor air circulation, guiding and assisting patients with liver cancer fever to drink more boiled water and change clothes frequently (keep clothes dry and clean). Strengthening oral care to prevent respiratory tract infection in patients with fever.
At the same time, we should pay attention to improving the immunity of patients with liver cancer fever, enhancing physical function and reducing infection and fever.
In the struggle between human beings and liver cancer, medical experts have devoted themselves to research and made great progress in treatment methods and drug research. Ginsenoside Rh2 is an important anti-tumor research achievement in recent years. Its high efficiency and non-toxicity make up for the shortcomings of some drugs and treatments in the past, and it will certainly play an important role in the field of anti-tumor.
Regulating the proliferation cycle of tumor cells and inducing apoptosis are the most basic and effective ways to inhibit tumors in the anti-tumor mechanism of ginsenoside Rh2.
In the regulation of proliferation cycle, Guo Zhiting of Jilin University and others have reported that ginsenoside Rh2 directly inhibits the DNA replication and transcription ability of cancer cells, and hinders the protein synthesis of cancer cells, thus slowing down or stopping the proliferation of tumor cells.
In the aspect of inducing apoptosis, it has been proved by experiments that many cyclin (cell cycle regulatory proteins) related to G 1 phase cell proliferation are regulated by ginsenoside Rh2, which leads to the growth stagnation of G 1 phase cells induced by ginsenoside Rh2, and finally leads to tumor cell apoptosis. AR (apoptosis rate) increased with the increase of ginsenoside Rh2 concentration and the extension of action time.
Zeng Xiaoli of Capital Medical University observed the effect of ginsenoside Rh2 on the proliferation, morphology and ultrastructure of human hepatocellular carcinoma cell SMMC-772 1, and found that ginsenoside Rh2 could inhibit the proliferation of SMMC-772 1 and induce the differentiation of SMMC-772 1. With the increase of concentration (effective concentration) and time, SMMC-772 1 hepatoma cells shrank, and the inhibition rate of SMMC-772 1 hepatoma cells increased, and there was an obvious dose-effect relationship.
Jiang Hao, the First Affiliated Hospital of University of South China, found that ginsenoside Rh2 inhibited the growth of human hepatocellular carcinoma cell Bel-7404 in a dose-dependent and time-dependent manner, indicating that ginsenoside Rh2 can inhibit the proliferation of hepatocellular carcinoma cell Bel-7404 and induce its apoptosis.
Hainan Asian Institute of Medicine is one of many Rh2 researchers. Not only the advanced technology has achieved great success in the extraction rate of Rh2, but also related products have been developed and entered the clinical application stage. In the process of clinical application, many successful cases show that ginsenoside Rh2 has the same significant anti-liver cancer effect, and many patients with liver cancer have obviously improved their symptoms and greatly improved their quality of life after taking this product.
With the wide application of ginsenoside Rh2, one patient after another with liver cancer has a long-lost but relieved smile. Liver cancer is rampant. Ginsenoside Rh2 challenges liver cancer with its remarkable curative effect and excellent quality. With the tenacious willpower and good mentality of patients with liver cancer, coupled with high-quality drugs and treatments, we have reason to believe that "cancer can be overcome and patients with liver cancer can enjoy a high-quality life more freely".
Mr. Zhong has liver cancer in his family.
Mr. Zhong's family, a native of Baoshan District, Shanghai, has advanced liver cancer. Because of age, other treatment methods were not considered. Now I just want to use drugs to control my illness and relieve the pain of the elderly. There is hepatic ascites, mainly by means of extraction. While taking ginsenoside Rh2, I am also taking some diuretics prescribed by the hospital. At present, ascites is basically controlled. Everything else is fine, but I don't want to eat. I only have occasional pains at night.
8- 16 Mr. Zhong bought three boxes of ginsenoside Rh2 for the elderly to take, and at that time he was also holding a try attitude. After taking it for a period of time, the appetite of the elderly has improved, and they will take the initiative to tell their families what food they want to eat. At that time, all his children were very happy. Mr. Zhong ordered three more cases. Up to now, appetite has improved and ascites has not recurred. There are still no other symptoms. Mr. Zhong thinks this is also a good phenomenon and hopes it can last for a long time. They are also very happy and will stick to it.
Liver cancer in Li Nainai
Mr. Li is from Shihezi, Xinjiang. 20 1 1 May, grandma was diagnosed with advanced liver cancer. She has pain, bloating, loss of appetite and can't walk. Because of her age and serious illness, the doctor gave up treatment. The family decided to take Chinese medicine. After learning about ginsenoside Rh2, they bought two boxes at the end of July. After taking it at the end of August, the condition remained stable. After that, they continued to take two boxes. At the end of September, the patient felt much better in pain and spirit. At that time, Mr. Li asked him to deliver two boxes every month for half a year.
65438+February, the patient's feedback effect is very good, and his appetite is much better than before. It used to hurt when walking, but now it doesn't hurt, and I don't have abdominal distension. My spirit is much better. In June+10, 5438, Mr. Li reported that his grandmother's condition was much better, which was not much different from that of normal old people. Considering the economic reasons, she temporarily reduced her consumption of some Chinese medicines.
Ginsenoside Rh2
[Chinese Name] 20(S)- Ginsenoside Rh2
[alias] 20(S)- ginsenoside Rh2, ginsenoside Rh2, ginsenoside Rh2
[English name] 20(S)- ginsenoside h2
[English alias] Ginsenoside 2,20 (s)-protopanaxadiol -3-o-β-d- glucopyranoside
[CAS] 67400- 17-3，782 14-33-2，88 156-29-0， 1 12246- 15-8， 1 17708-9 1-5
[Molecular weight] 622.88
[purity] HPLC > 99%
Ginsenoside Rh2 is a protopanaxadiol-type low-sugar chain saponin monomer separated from ginseng. It is a secondary saponin produced by the thermal decomposition of some protopanaxadiol ginsenoside when raw ginseng is processed into red ginseng. Ginsenoside Rh2, the "gold" component of ginseng, has a strong anti-tumor effect and a synergistic effect on traditional chemotherapy, which can reduce the toxic and side effects of chemotherapy and improve the immunity of the body. After some clinical research and practice, it is considered to be the most potential natural anticancer Chinese medicine.
Ginsenoside Rh2 can inhibit tumor invasion and metastasis, induce cancer cell apoptosis and inhibit tumor angiogenesis by regulating immune function. Reversing the drug resistance of tumor cells and enhancing the efficacy of anticancer drugs; Induce cancer cell differentiation and inhibit cancer cell growth; It also has the functions of antagonizing carcinogens and chemically preventing cancer.
Studies have proved that taking ginsenoside Rh2 for a long time can increase resistance, shrink tumors, reduce the harm of radiotherapy and chemotherapy, prevent complications such as jaundice, pain, ascites, edema and hoarseness, and also improve the quality of life of patients with advanced cancer and prolong their lives.
Ginsenoside Rh2 can play an anti-tumor role by regulating the proliferation cycle of tumor cells, inducing cell differentiation and apoptosis, and has a wide range of anti-tumor activities on a variety of tumor cells. Combined application with western medicine can reduce the dosage of western medicine, reduce side effects and enhance patient compliance. In addition, ginsenoside Rh2 is safe and has no obvious effect on nervous system, respiratory system and cardiovascular system, which is obviously different from other anticancer chemotherapy drugs. Ginsenoside Rh2, as a natural anti-tumor component, will effectively prevent and treat tumor diseases, relieve pain for cancer patients, prevent cancer, maintain cancer and make cancer rehabilitation possible.
Research Progress on Anti-tumor Mechanism of Ginsenoside Rh2
Guo Zhiting, Shao, Fu Wenwen, Wei
(College of Animal Husbandry and Veterinary Medicine, Jilin University, Changchun, Jilin 130062)
The research progress of anti-tumor mechanism of ginsenoside -Rh2 (G-Rh2) extracted from ginseng was discussed. The anti-tumor activity and its mechanism of G-Rh2 in regulating the signal pathway system of tumor cells, reversing the abnormal differentiation and drug resistance of tumor cells, reducing telomerase activity of tumor cells, enhancing immunity, improving anti-tumor ability and blocking the synthesis and metabolism of some important components of tumor cells were discussed.
Key words: ginsenoside-rh2; Anti-tumor; telomerase
China classification number: S 853.74 Document ID: A document number:1000-6419 (2005) 09-0742-04.
Tumor is a serious threat to human and animal health and even life-threatening disease. In the literature of traditional Chinese medicine, the record of Chinese herbal medicine in preventing and treating tumors has a long history. As a traditional Chinese herbal medicine, ginseng was used to prevent and treat diseases as early as 2000 years ago. At present, more than 40 kinds of ginsenoside monomer components have been isolated from ginseng, among which ginsenoside 2H2 (G-Rh2) is one of the natural active components extracted from ginseng. Studies have confirmed that this component has strong anti-tumor activity. In recent years, the anti-tumor effect of G2Rh2 has attracted the attention of researchers at home and abroad. The mechanism of anti-tumor effect of G-Rh2 was reviewed.
1 regulates tumor cell signal pathway system.
Cell signaling pathway system means that cells receive external signals and convert extracellular signals into intracellular signals through a set of specific mechanisms, and finally regulate the expression of specific genes and cause cell reactions. The study of signal system is the frontier of cell biology, so it is of great significance to study the anti-tumor mechanism of G2Rh2 from the perspective of cell signal pathway.
1. 1 protein kinase C( PKC), insulin-like growth factor (IG2 Fs) and insulin-like growth factor I receptor (IGF IR) signaling pathways
P KC is a Ca2+/ phospholipid-dependent ser/ Thr protein kinase, which participates in signal transduction of various physiological functions of cells and is considered as a "classic signal transmitter" for many extracellular stimuli to induce the production of multiple lipid second messengers. Zeng Xiaoli and others reported that G-Rh2 may inhibit the translocation and activation of P KC by decreasing the intracellular Ca2+ concentration, and ultimately hinder the proliferation signal transduction process mediated by P KCα by inhibiting the expression of P KCα protein in cells.
For many malignant tumor cells, exogenous and autocrine IGF are strong mitogenic agents. Signal transduction mediated by IGFs and IGF IR can not only promote the proliferation of malignant tumor cells, but also play a key role in cell transformation and tumor occurrence and development. It is suggested that G-Rh2 may block the signal transduction pathway of IGFS-IGF-IR proliferation by reducing the expression of IGF-IR in hepatocellular carcinoma cells, thus inhibiting the proliferation of hepatocellular carcinoma cells and inducing their differentiation. In addition, in the process of G-Rh2-induced apoptosis of human fibroblasts SK-N-BE (2) cells, with the extension of treatment time, the three types of P KC-α, β and γ-gradually increased, while in C6Bu- 1 cells, the changes of P KC of α, γ, δ and ε types were not obvious. The results suggest that cells with or without P-KC type may induce apoptosis through different bypass pathways and interactions under different G-Rh2 effects.
1.2Caspase and Bcl-2 signaling pathways
Caspase protein family is a group of proteases related to cytokine maturation and apoptosis, which plays an important role in signal transduction of tumors. By regulating its expression level in tumor cells, the cure rate of tumor can be effectively improved. At the early stage of G-Rh2 acting on SK-N- BE(2), the amount of p53 rapidly accumulated, which promoted cell apoptosis. According to the results, it is speculated that the activation of Caspase- 1 and Caspase-3 and the up-regulation of Bax participate in the apoptosis induced by G-Rh2. G-rh2 can also inhibit the proliferation of A375-S2 cells in vitro by inducing apoptosis, and the apoptosis induced by G-Rh2 partly depends on the bypass pathway of Caspase-8 and Caspase-3.
Bcl-2, as a signal transduction substance, can inhibit various forms of apoptosis, mainly by increasing the permeability of mitochondrial membrane, so that cell growth stays in G0 phase, thus achieving the purpose of mediating the signal pathway of tumor cells. A series of characteristic morphological changes of apoptotic cells, such as cell atrophy, chromatin concentration and nuclear concentration, were observed in rat glioma C6Bu- 1 cells treated with G-Rh2. However, the author thinks that apoptosis induced by G-RH2 is not through the bypass pathway of Bcl-2, Bcl-xL and Bax, but through other pathways. When studying the effect of G-Rh2 on SK-H EP- 1 cells, Jin et al. found that the activity of cyclin A-Cdk2 increased sharply, which was blocked by the compound expression of a dominant negative allele of Cdk2, thus promoting cell apoptosis. Some people think that G-Rh2 induces apoptosis by producing reactive oxygen species (ROS).
G-Rh2 achieves the anti-tumor effect mainly by reducing the intracellular P KC level and mediating the signal pathways such as Caspase and Bcl-2. However, this regulatory effect is often not exerted by a single channel, but by the interaction of multiple channels, but the specific mechanism is still unclear and needs further study and confirmation.
2 affect the activity of telomerase in cells
Telomerase is an RNA reverse transcriptase, which can reverse transcribe and synthesize important sequences of telomeres using its own RNA as a template. Telomerase is a specific marker of tumor. Normal somatic cells are negative for telomerase, while most tumor cells are positive. The detection of telomerase is helpful for the diagnosis of tumor, judging the malignant degree and prognosis of malignant tumor, and can also be used as an evaluation index to measure the effect of various anti-tumor treatments. It has been found that G-Rh2 can induce the differentiation of 772 1 hepatoma cell line in vitro, and G-Rh2 can promote the cell differentiation to normal state by reversing the transcription level of transcriptase mRNA by telomerase, and effectively inhibit the activity of telomerase. This inhibitory effect is time-dependent: at 1. After 105 treatment, its activity decreased to 0.765 (P
Blocking the synthesis and metabolism of important components in tumor cells
Studies have confirmed that blocking the synthesis or inhibiting the expression of some important components in tumor cells is an effective anti-tumor approach in the process of preventing and treating tumors. Jun N-terminal kinase 1(JNK 1) is one of the important components of cells, which can obviously promote the apoptosis of tumor cells. Ham et al. reported that in the process of G-Rh2 inducing apoptosis of SK-H EP- 1, the activity of JN K 1 was rapidly increased and prolonged, while JBD, an inhibitory protein expressed as a dominant negative mutant -DN-JN K 1 or JN K 1, was obviously inhibited. In addition, the activation of JN K 1 mediated by G-Rh2 is related to the activity of SAPK/ ERK kinase 1 (SEK 10~30 minutes, the first 10~30 minutes, but the subsequent continuous activation is related to SEK/kloc-0. It can be seen that SE K 1 plays a very important role in the activation of JN K 1, but there is no simple correlation. G-Rh2 can also reduce the level of cyclin D by inducing the protein expression of p2 1, thus down-regulating the activity of cyclin/Cdk complex kinase, weakening the phosphorylation of retinal glioma protein (p Rb) and inhibiting the release of E2 F, thus inhibiting the proliferation of MCF-7 cells. Phospholipase C plays an important role in the anti-tumor process and is necessary for the activation of P KC. G-Rh2 can effectively reduce the activity of phospholipase C, and as a result, it can reduce the level of endogenous activator of P KC in cells, thus playing an anti-proliferation role. Hyun-Eui et al. [16] found that after G-Rh2 treatment, the specific polymerase (ADP- ribose) did not break into 85 ku protein as in other apoptosis experiments, and the expression levels of Bcl-2 and Bax did not change. Over-expressed antioxidant protein Bcl-Xl is also similar to that of parent cells. It has been proved that c-myc is an oncogene, and the amplification or high expression of c-myc gene has been found in many human tumor tissues. It is reported that G-Rh2 can also reduce the expression of c-myc gene. The above results show that G-Rh2 plays an anti-tumor role mainly by blocking the synthesis and metabolism of some important enzymes in the biological reaction system. In general, the treatment effect of such malignant tumors is thorough and it is not easy to recur.
Reversing abnormal differentiation and drug resistance of tumor cells
4. 1 Reverses the abnormal differentiation of tumor cells.
Because the abnormal differentiation of tumor cells plays an important role in the pathogenesis of tumors, some scholars have proposed that drugs should be used to reverse the abnormal differentiation of tumor cells, that is, to induce tumor cells to differentiate into normal cells or nearly normal cells. As a new way to study anti-tumor drugs, it is a hot research topic to find differentiation inducers from natural compounds. It was found that G-Rh2 could induce human promyelocytic leukemia HL60 cells to differentiate into normal granulocytes, and the cells were arrested in G 1/ S phase, which had a strong induction effect. P KC participated in this process. It is generally believed that morphological differentiation and maturity is a sign of malignant tumor cell differentiation. Morphological differentiation of SMMC-772 1 cells by 20μg/ mL G-Rh2 confirmed that the development state of organelles changed, and its morphological structure obviously changed to the normal direction, and the production of alpha-fetoprotein (AFP), γ -glutamyltranspeptidase (γ-GT) and heat-resistant alkaline phosphatase (ALP) decreased significantly. 0.0 1) 。 Morphologically, G-Rh2 induces tumor cells to transform into normal cells. Zhou Guihua and others found that G-Rh2 had a strong differentiation induction effect on mouse melanoma B 16 cells at the concentration of 10 mg/L, which showed that the melanogenesis ability was obviously enhanced and the cells differentiated into normal epithelioid cells. If PC-3M cells were incubated with different concentrations of G-Rh2 *** for 24 h, it was found that G-Rh2 could obviously inhibit the growth of PC-3M cells in vitro in a concentration-dependent manner, and the maximum inhibition rate could reach 865,438 0.7%, and the cell morphology became normal. In clinical treatment, it is of great significance to improve the curative effect if cancer cells can be diagnosed early and reversed in time.
4.2 Reversing tumor drug resistance
In the process of treating tumor, it is found that the failure of clinical chemotherapy is often related to the drug resistance of tumor cells. Studies have proved that G-Rh2 can be used as a drug resistance reversal agent to improve the anti-tumor activity of chemotherapy drugs. G-Rh2 can promote the reversal of drug resistance of leukemia drug-resistant cell line P388/ ADM in vitro, thus enhancing the cytotoxicity of mucomycin and vinblastine.
It is generally believed that G-Rh2 plays two roles in chemotherapy drugs for cancer cells, that is, additive or synergistic. It is reported that it can sensitize a variety of breast cancer cells resistant to violet alcohol. G-Rh2 has strong anti-tumor activity, and can be used to treat cancers resistant to various anti-tumor drugs, especially when combined with traditional chemotherapy drugs. When the dose is 15. When 0 mg/ L of G-Rh2 was combined with 0.4 mg/L of cisplatin (DDP), the inhibition rate of PC-3M was the same as that of 10 times DDP (4. 0 mg/ L), indicating that G-Rh2 can promote PC-3M. The mechanism may be that the two drugs induce apoptosis of tumor cells through different ways, resulting in synergistic effect, thus improving the ability of low-dose DDP to induce apoptosis.
5. Enhance immunity and improve anti-tumor ability.
G-Rh2 can improve the immunity of the body, so as to achieve the effect of inhibiting cancer, which is also a major feature of its superiority over other anticancer drugs. As an immunomodulatory factor, G-Rh2 can promote the synthesis of albumin and γ-globulin, improve the functions of T cells and macrophages, thus inhibiting the proliferation of tumor cells, and at the same time inhibiting the chromosomal mutation of normal lymphocytes, stabilizing the immune system and achieving the effect of inhibiting cancer. Studies have confirmed that G-Rh2 injection can significantly improve the phagocytic function of peritoneal macrophages in tumor-bearing mice and promote tumor-bearing.
Bone cancer is rarely found by accident, and it is usually found late, so families with conditions or genetic predisposition can start prevention in advance, but it is most important for patients who have been found in treatment and rehabilitation to maintain a good attitude. If you have a good attitude, you will get better treatment. . . .
Effect of ginsenoside Rh2
1. regulates the cell proliferation cycle and mainly kills tumor cells in the proliferative phase.
2. Induce tumor cell apoptosis without inflammation.
3. Adjuvant chemotherapy to enhance the sensitivity of chemotherapy drugs.
4. Reversing the drug resistance of tumor and enhancing the curative effect of traditional chemotherapy.
5. Reduce the side effects of chemotherapy and increase the number of white blood cells.
6. Induce tumor cells to differentiate into normal cells
7. Prevent tumor metastasis
8. Prevent tumor recurrence in recovery period.
9. Balance the immune system and prevent cancer.
10. Multiple anti-fatigue effects, quickly recovering fatigue induced by physiological or psychological factors, effectively relieving dizziness, drowsiness and other symptoms, and increasing the consumption of fatty acids, thus generating energy, invigorating the spirit, improving endurance and improving physical fitness.
Ginsenoside Rh2 prevents cancer recurrence and metastasis.
Tumors seriously endanger human health. At present, surgery, radiotherapy, chemotherapy and biological methods are mainly used. However, most drugs are cytotoxic and lack specificity. When killing tumor cells, it often kills normal cells indiscriminately, especially hematopoietic stem cells with active proliferation. The damage of normal cells often leads to serious complications, which affects the therapeutic effect and patient compliance and greatly reduces the patient's own immunity.
Cancer has the characteristics of high recurrence and metastasis rate. Recurrent cancer refers to the growth of new tumors on the organs where the primary cancer is located after the primary cancer has subsided after treatment. Metastatic cancer is that cancer cells invade blood vessels, lymphatic vessels or body cavities from the primary site, run with blood or body fluids, and form the same type of cancer as the primary cancer in distant sites or organs. Therefore, after surgery, radiotherapy and chemotherapy, it is often not once and for all, and the possibility of recurrence and metastasis is greater. There are many factors that cause recurrence and metastasis, among which low immunity is a key factor. As we said before, simultaneous operation, radiotherapy and chemotherapy will greatly harm the immunity of patients. While ginsenoside Rh2 is different.
Ginsenoside Rh2 enhances the immunity and anti-tumor ability, thus achieving anti-cancer effect, which is also a major feature of its superiority over other anti-cancer drugs. Ginsenoside Rh2, as an immunomodulatory factor, can promote the synthesis of albumin and γ-globulin, improve the functions of T cells and macrophages, thus inhibiting the proliferation of tumor cells, and at the same time inhibiting the chromosomal mutation of normal lymphocytes, stabilizing the immune system and achieving the effect of inhibiting cancer. Studies have confirmed that ginsenoside Rh2 injection can significantly improve the phagocytic function of peritoneal macrophages in tumor-bearing mice and promote the production of erythrocyte antibodies in serum of tumor-bearing mice. For example, when the activity of NK cells in mice with liver cancer (H22) model group is seriously low, the killing activity of high and medium doses of ginsenoside Rh2 on NK cells is obviously enhanced, and the activity of IL-2 is significantly improved in high dose group of ginsenoside Rh2. It has also been reported that ginsenoside Rh2 and panaxadiol 20-O-β-D- glucopyranose -sy-20(s)- panaxadiol metabolized by intestinal bacteria can block the metastasis of tumor cells to extracellular matrix and basement membrane by inducing immunomodulation mechanism.
The above research shows that ginsenoside Rh2 can play an anti-tumor role by regulating and enhancing the immune function of the body, and has a wide range of anti-tumor activities on a variety of tumor cells. If ginsenoside Rh2 is combined with western medicine, the dosage of western medicine can be reduced, the side effects can be reduced, and the compliance of patients can be enhanced. Kill two birds with one stone by improving immunity while treating cancer.
Functional energy can improve autoimmune function and enhance disease resistance; It is a good auxiliary health food for patients with pancreatic cancer, breast cancer, ovarian cancer, liver cancer, lung cancer, gastric cancer, lymphoma, nasopharyngeal cancer, glioma, bone cancer, cervical cancer, colon cancer, melanoma, leukemia, esophageal cancer, prostate cancer and other cancers in the process of treatment and rehabilitation.
The right person is mainly suitable for the following situations:
1. Patients who have just been diagnosed with cancer, but it is still difficult to determine the specific treatment plan, can control the development of the disease and prevent tumor metastasis and spread after taking it;
2. Patients taking it before operation can shrink the tumor, narrow the scope of operation and improve the success rate of operation;
3. After the operation, the patient can eliminate the residual cancer cells, prevent recurrence and metastasis, and at the same time accelerate the healing of the incision, diminish inflammation and relieve pain, prevent infection and prevent complications;
4. Patients with radiotherapy and chemotherapy can eliminate toxic and side effects, enhance drug sensitivity and improve curative effect;
5. Patients with acute, severe and advanced cancer are weak. Taking ginsenoside Rh2 can improve autoimmune function, enhance disease resistance, improve quality of life and prolong life.
6. Patients in cancer recovery period can take it regularly, which can effectively eliminate the residual tumor cells and minor lesions in the body, enhance their physique, consolidate their curative effect and prevent recurrence and metastasis.
7. People with a family history of cancer
8. People with low immunity
Not suitable for people/kloc-children under 0/4 years old
Advanced patented technology and reasonable price guarantee
The content of ginsenoside Rh2 in ginseng is very small, and the extraction rate of ginsenoside Rh2 is only1100000 by conventional extraction technology.
The company adopts patented technology, which can improve the extraction rate by nearly a thousand times, greatly improve the production efficiency and reduce the production cost, and provide reliable guarantee for setting reasonable prices and reducing the use cost of patients.
Comprehensive action to ensure the effect
Scientific experiments have proved that ginsenoside Rh2 not only has the anti-cancer and anti-cancer effects of inhibiting cancer cell proliferation, inhibiting carcinogenic activity, inducing and inducing cancer cell differentiation and apoptosis, resisting tumor invasion and metastasis, and reversing tumor drug resistance, but also can improve the chemotherapy effect, overcome the toxic and side effects of chemotherapy, enhance the body's immune function, and comprehensively restore the patient's physique.
Efficient, non-toxic and painless
The main methods of tumor treatment are surgery, radiotherapy and chemotherapy. Chemotherapy and radiotherapy not only kill tumor cells by radiation and chemical drugs, but also kill normal cells, especially hematopoietic cells, which often lead to decreased immunity, low nutritional status, infection and even other complications. Therefore, patients often have symptoms such as nausea, vomiting, alopecia, loss of appetite, thrombocytopenia and leukopenia, and extreme weight loss while undergoing routine radiotherapy and chemotherapy. Taking ginsenoside Rh2 can increase patients' appetite, make the number of platelets and white blood cells return to normal, and there are few adverse symptoms such as nausea and vomiting. At the same time, it also has good analgesic and sleep-helping effects, improves the quality of life of patients, and enables patients to maintain normal physical strength all the time and successfully complete various treatments.