Deafness and tinnitus caused by Meniere's disease, who can help me? I really can't bear it anymore! Patient's age: 22 Patient's gender: Female All symptoms: Deafness and tinnitus may be caused by Meniere's disease Time and cause of onset: 2003, the cause is unknown Treatment status: The doctor's judgment during previous treatment may be too one-sided and no specific cause was found . I went to many hospitals and they all said it was neurological deafness. I want help: clarify the cause and cure the disease. Condition analysis: Hello, the causes of tinnitus and deafness are very complicated, and the nature of tinnitus is also different, such as the sound of cicadas, The sound of wind, train, etc., for different natures of tinnitus and different causes of tinnitus, the treatment plans will be different. Opinions and suggestions: There are many methods that can be used to treat tinnitus and deafness, including interventional therapy, audio frequency vibration, negative pressure oxygen therapy, and Chinese herbal fumigation. Among them, Hancao fumigation is a nationally patented method for treating tinnitus and deafness, and the clinical effect is very significant. The specific method to take still needs to be selected according to your condition. If Chinese herbal fumigation is used for treatment, it is recommended that a course of treatment last for 10 days. After a course of treatment, obvious effects can basically be observed, and the treatment time and plan can be adjusted as appropriate based on the situation after treatment. Generally, blood vessel dilating drugs are used for general treatment. You can try Ginkgo biloba leaves and Nuodikang capsules. You can also cooperate with acupuncture, massage and other methods of treatment. I wish you a speedy recovery! Symptoms of Meniere's disease Meniere's disease is a continuous pathophysiological process, which can be caused by the blockage of the pathway of endolymph fluid reaching the endolymphatic sac, or the endolymphatic sac and ducts of endolymph reabsorption. Pathological changes in the endolymph cause the regulatory mechanism of the endolymphatic microfluid environment to be destroyed and promote the progressive accumulation of endolymph fluid. In contrast, latent endolymphatic hydrops is mostly discovered during postmortem temporal bone anatomy, and mostly occurs around the top of the cochlea in humans or animals. Symptoms can only occur in three cases, namely endolymphatic hydrops. No further development, combined with degeneration of sensory nerve structures or spontaneous membranous labyrinth fistula formation. The pathological characteristics of progressive endolymphatic hydrops include the following aspects: 1. Endolymphatic hydrops: In membranous labyrinth hydrops, the expansion area of ??the vestibular membrane and saccule wall is the largest, which is easily observed and is more oval. The sac and semicircular canals are more susceptible to dilatation. However, severe cases may be accompanied by expansion of the entire endolymphatic system, causing the vestibular membrane to adhere closely to the bone wall of the scala vestibuli, or even swell into the cochlear foramen. The wall of the saccule can expand to the footplate of the stapes, and the utricle and the ampulla of the semicircular canals may also expand and deform. 2. Rupture of the membranous labyrinth: It has been proven that any part of the membranous labyrinth may be ruptured and distorted, but only the semicircular canals The non-ampullary is rare. Even though a slight slow leak of electrolytes can be observed at the submicroscopic level, implicating sudden and widespread potassium intoxication as the cause of vestibular and cochlear symptoms, there must be healing of the membranous structures between the two ruptures. Thus, recurrent episodes of vertigo and fluctuating hearing loss may be clinical manifestations of this breakdown-healing cycle. 3. Labyrinth fistula formation: There is enough evidence to show that a large area of ??rupture in the membranous labyrinth may cause a persistent fistula that cannot be healed, making it impossible for further expansion and rupture of the labyrinth lesion. It has been observed that progressive The membranous expansion and distortion of endolymphatic hydrops can hinder the longitudinal flow of endolymph fluid. This obstruction can occur in the sinuses of collecting tubules, saccule ducts, utricular ducts, or endolymphatic vessels. In this condition, one part of the endolymphatic system forms a fistula, while another part undergoes repeated dilation and rupture. The coexistence of cochlear fistulas and balloon wall collapse can even be observed. On the other hand, ruptures of the utricle and ampulla of the semicircular canals often show signs of healing. During the onset of Meniere's disease, the formation of fistulas in the cochlear duct, saccule, and utricle is often seen, which explains the natural remission of vestibular symptoms in most patients with Meniere's disease. 4. Vestibular fibrosis: Many cases of progressive endolymphatic hydrops are accompanied by fibrous tissue hyperplasia in the vestibule. Because the saccule, utricle, or cochlear duct conflict with each other, they may rupture each other or become thickened due to fibrous tissue proliferation, eventually forming irregular fibrous strips across the vestibule from the membranous labyrinth to the stapes footplate. When the footplate moves, Causes dizziness and nystagmus (Hennebert's sign), accounting for about 35%.
Therefore, Hennebert's sign (a positive fistula test in a normal ear or bony labyrinth) strongly suggests the presence of long-standing endolymphatic hydrops. 5. Sensory system lesions: In most cases of Meniere's disease, there is no obvious loss of hair cell groups in the organ of Corti or vestibular receptors, and there is not even severe sensory dysfunction. At present, we do not know whether the functional changes in this disease are caused by changes in the biochemical environment of the fine body fluids of the inner ear, changes in sensory and cognitive mechanisms, changes in biopotentials, or the combined effect of these factors. However, it is certain that these clinical features cannot be fully explained by simple functional deficits of sensory or neural units alone. During the developmental stages of Meniere's disease, there are persistent morphological changes in the sensory organs. Changes in the organ of Corti include loss of hair cells, atrophy of supporting cells, deformation and atrophy of the tectorial membrane, and sinking of the vestibular membrane into the organ of Corti. In the vestibular system, significant distortion of the ampullar wall of the semicircular canals can affect the movement of the cristae cap. Due to the extremely enlarged saccule, the utricle macula prolapses into the upper wall of the vestibule, causing changes in the spatial positioning of the utricle macula. These utricular changes may explain the dumping sensation experienced in these patients. 6. Nerve damage: About 10% of ears with Meniere's disease have loss of cochlear neurogen, and a small number of cochlear periapical hair cells disappear. When the potassium-contaminated perilymph reaches the scala tympani through the cochlear foramen, it may change the electrolyte environment of the peripheral nerve fibers in this area, causing cell atrophy. Another common histological finding in cochlear specimens with human endolymphatic hydrops is the herniation of the vestibular membrane through the cochlear foramen into the periparietal scala tympani. It is hypothesized that this condition causes obstruction of the canaliculi, thereby altering the periparietal nerve fibers and The tissue fluid environment of the organ of Corti. During this time, loss of vestibular system neurons is rarely observed. Even if there are severe changes in the sensory organs, these nerve fibers and neuron groups remain intact. As we know, the vestibular nerve is less likely to atrophy than the cochlear nerve after sensory organ damage and also ages more slowly. Recommended therapy for the treatment of deafness and tinnitus: "Argon iontophoresis therapy" gives you a world of sound. "Argon iontophoresis therapy" used by the Department of Otolaryngology. Since its clinical application, this therapy has relieved the pain of deafness, tinnitus and tinnitus patients across the country and enabled them to get out of trouble. Silent world. Compared with other therapies for treating deafness and tinnitus, this therapy has incomparable advantages and efficacy in clearing ear toxins, opening ear orifices, and fully activating ear nerves. It is specifically reflected in several aspects: First, the unique effect of clearing ear toxins The nano-active ingredient, the ear-clearing factor, reaches the cochlea along the meridian channels: softening blood vessels, dissolving and expelling toxins deposited in the ear veins, the ear veins are dredged, the blood circulation in the ear is improved, the blood flow is accelerated, the oxygen carrying capacity of the blood is increased, and the speed is accelerated. The metabolism of ear nerves can comprehensively improve the pathological environment of auditory nerve damage. Second, open the ear orifices. Through this therapy, the meridians of the ear orifices are opened, the meridians of the ear orifices are connected, the meridians are smooth, the yin and yang qi and blood flow, the clear and yang qi can flow upward, and the external air can enter the interior, nourishing the ear nerves and improving immunity. It promotes the growth and expansion of ear nerves and brings a large number of ear nerves back from the edge of death. Third, activate the ear nerve, which specifically activates the mitochondrial enzyme of the hair cells, releases enough energy to excite the hair cells, converts sound into bioelectric energy, activates the ear nerve, restores hearing function, and enables the deaf-mute to completely step out of the silent world. . This therapy has been clinically verified by more than 30 authoritative institutions such as the Chinese Academy of Medical Sciences’ Ear Disease Research Center and the Chinese Medical Association. It can treat various tinnitus, neurological deafness, tinnitus, drug-induced deafness, tinnitus, noise-induced deafness, and sudden deafness. The effect is remarkable on tinnitus, acute and chronic otitis media, hearing loss and dizziness, and Meniere's syndrome! It brings various ear diseases such as deafness, tinnitus, and tinnitus into the latest realm of fundamental cure, overall recovery, and treatment of both symptoms and root causes.