This disease is a skin disease characterized by bright red or pale white air masses. Because it is as small as a bean and as big as a block, it is caused by wind every time, so it is also called "rash".
This disease was recorded as early as Su Wen's "Four-time Punctuation Violation Theory", saying: "Less yin is more than enough, skin is blocked, and rash is insufficient". Since then, there have been many records in the literature of past dynasties, such as the article "synopsis of the golden chamber" Stroke calendar ",which says:" After the evil, the body itches and rashes. " It can be seen that the ancients knew this disease for a long time.
Its etiology and pathogenesis are always caused by wind evil. For example, The Theory of Staging of Pathogens says: "People's skin is weak, and if it is broken by wind, there will be hidden rash." But the wind is the length of all diseases, and other diseases are often attached to the wind and cause diseases. Clinically, wind and cold or heat are combined and depressed in the skin, resulting in this disease, but there are differences between exogenous and endogenous.
1. Exogenous factors: pathogenic factors of wind-cold or wind-heat occur between fur. For example, the conclusion of pathogenic qi depression points out: "pathogenic qi exists in the skin, and when the cold breaks, the wind will scratch out a rash." "Madam Yang deficiency leads to excessive sweating, and sweating is wind, and wind beats hot air, so that N is prepared", which clearly points out that wind-cold and wind-heat invade the skin, or superficial deficiency is not solid, and wind evil takes advantage of deficiency and causes disease.
2. Endogenous: Overeating greasy, thick-smelling, fishy and other things that help fire and wind, such as fish, shrimp and crabs. , or because of intestinal worms, can lead to gastrointestinal disharmony, endogenous wind and fire, depressed in the skin. If the patient is ill for a long time, qi and blood will be consumed, so that blood deficiency will lead to wind (women will not be able to adjust their position) and wind evil will occur in the skin.
Clinical manifestations: First, acute attack, frequent eating and nausea, such as fish, shrimp, crabs and other seafood, or taking certain drugs, or catching a cold, or having a history of intestinal parasites.
Second, the rash varies in size and shape, with bright red or yellowish air mass, clear boundary and severe itching.
Third, skin pain often occurs suddenly, and gradually subsides after several minutes to several hours. Later, it appeared in batches, appeared and disappeared, leaving no trace. Wholesale in batches or in batches every day, and it will not be distributed in a week or so. Chronic patients have recurrent attacks for weeks, months or even years.
Four, some cases may be accompanied by diarrhea, abdominal pain, nausea and vomiting and other intestinal symptoms. When it affects the throat, it may cause throat discomfort and hoarseness, and even laryngeal edema, resulting in difficulty breathing and even suffocation. Some people may have positive skin scratches.
5. The rash is found on the lips and eyelids, and then the skin color is reddish and the swelling boundary is unclear. This rash is the facial wind when wandering on the lips and face (called vascular edema in western medicine). If normal skin is scratched mechanically, scratching-like edema and swelling will appear in this part, which is called artificial urticaria or skin scratching.
Clinical classification: 1. Wind-heat type: the wind mass is red, which occurs when it is hot, decreases when it is cold, and recovers in summer or is heavy in summer and light in winter. Thin yellow tongue coating and floating pulse.
Second, wind-cold syndrome: the wind mass is white, and when it is cold outside or cold wind blows, it gradually warms up and decreases, and it is warm in winter or heavy in summer, with thin white tongue coating and tight pulse.
3. Gastrointestinal damp-heat syndrome: The onset may be accompanied by abdominal pain or periumbilical pain, mental fatigue, constipation or diarrhea, or nausea and vomiting. The tongue coating is yellow and greasy and the pulse is slippery.
4. Syndrome of deficiency of both qi and blood: recurrent wind mass, internal heat and external cold, frequent occurrence in autumn and winter, red papules, generalized convulsions, etc. It lasts for several months or years, and then it attacks or aggravates after fatigue, which makes the mind tired. The tongue is pale, the coating is thin and white, and the pulse is thin or heavy. (If a woman has irregular menstruation, she often begins to have wheal a few days before menstrual cramps, and then disappears. The process is periodic and may be accompanied by menstrual pain or irregular menstruation. Its tongue coating is thin and light, with thin pulse or thin string.
Differentiation: 1. Erythema multiforme, which mostly occurs in the exposed parts such as the back of the hand and the back of the foot, is a round papule or a round maculopapule with an iris-like center and a lasting rash.
Second, papular urticaria: the damage is spindle-shaped edematous papules with blisters with large needles at the top. Rash lasts for a long time, leaving temporary pigmentation after it subsides, and it usually occurs in lower limbs and trunk, mostly in spring and autumn. Children are more common. 3. Names of TCM clinical diseases, etc.
Treatment: divided into internal treatment and external treatment. Add and subtract prescriptions according to each syndrome type; That is, according to the clinical manifestations and symptoms of the patient's detailed condition, dialectical medication can be effective.
Examples of urticaria:
Formula for treating urticaria
I had urticaria when I was young. I introduce as follows: 5 cicadas, 250 grams of duckweed in the pond, decocted in water twice a day (once in the morning and once in the evening). Results It was cured after 3 doses.
Fresh towel gourd leaves can cure urticaria.
I went to primary school for five or six years ... >>
Question 2: How to completely cure itchy urticaria? It's disgusting. You can use Mimiyou plant antipruritic spray, which can immediately relieve itching and reduce swelling. Details of ........... can be found online.
Question 3: How to treat measles by scratching? You mean scratching urticaria, right?
Scrub the whole body with boiled water from old pea seedlings and wild radish seedlings, and wash it several times.
Don't worry too much.
Question 4: How to treat chronic urticaria scratching? This should be a chronic skin scratch, which belongs to urticaria.
Drug treatment is difficult to be effective, only pay more attention to life and diet.
Eat more fresh fruits and vegetables rich in vitamins or take vitamins C and B, or B 6 in group B..
Eat more alkaline foods, such as grapes, green tea, kelp, tomatoes, sesame seeds, cucumbers, carrots, bananas, apples, oranges, radishes, mung beans and nuts. Rest more, don't be tired, and exercise moderately. All these contribute to the natural mechanism of allergens in the body.
Avoid spicy and warm food: spicy and warm food will harm the body and often lead to recurrence. Like wine, strong tea, coffee,
Pepper, garlic, leek, dog meat, sparrow meat, shrimp, etc. Not edible. In addition, it belongs to sweets, such as mutton, chicken, pumpkin,
Taro, longan, chestnuts, carp and silver carp should also be eaten less.
Now there is another folk prescription: take vitamin C calcium tablets, and if necessary, you can order strong pine tablets, which really works. I hope you get well soon!
Please don't copy the special answer of heart blade! thank you
Question 5: Where is the treatment and repair of scratch urticaria better?
New discovery of chronic urticaria: Kangminyuan antiallergic probiotics reduce urticaria allergen-specific IgE
Chronic urticaria is a common skin disease, characterized by itching and wheal, which is usually induced by allergic and non-allergic mechanisms. About 15%~25% of patients have suffered from urticaria in their lifetime, and 80%~90% of urticaria can't find external causes. These patients are traditionally called chronic idiopathic urticaria. The average course of chronic urticaria is 3~5 years, and some patients can last for 20 years.
Disease classification of chronic urticaria
The classification of diseases is to better understand its pathogenesis and guide treatment. According to the medical history and laboratory examination, urticaria can be divided into allergic urticaria and non-allergic urticaria, in which allergic urticaria can be divided into allergen-specific IgE-mediated urticaria and non-IgE-mediated urticaria. The purpose of this in-depth classification is to try specific immunotherapy to treat IgE-mediated urticaria. The infiltration mode of inflammatory cells in chronic urticaria is similar to the delayed phase reaction induced by allergens, showing superficial perivascular inflammation. However, cytokine secretion patterns {Interleukin -4(IL-4), Interleukin -5(IL-5) and r-IFN)} indicate a Th0 or mixed th6+0/Th2 response pattern, rather than a simple Th2 response.
Frontier science of anti-allergic specific IgE antibody in the treatment of chronic urticaria;
At present, the pathogenesis of chronic urticaria is mostly studied by autoimmune theory, and the incidence of autoimmune urticaria has been paid more and more attention by clinical urticaria research. Although histamine is considered to be the most important mediator in the pathogenesis of urticaria, patients with acute and chronic urticaria will soon disappear after taking antihistamines, and urticaria will also be relieved. According to the above IgE-mediated immune theory, mast cells in tissues and basophils circulating or chemotactic to tissues are the two main sources of histamine production, while there are IgE high affinity receptors on the surfaces of mast cells and basophils, and allergen binding can lead to the cross-linking of the latter and induce the release of histamine, the main inflammatory mediator in cells. Histamine and other mediators can also be released through non-allergic mechanisms. Besides histamine, other mast cell mediators (alopecia) also play a role in urticaria.
Some studies believe that about two-thirds of chronic urticaria is related to autoimmunity, and there is sufficient evidence to prove that some patients with chronic urticaria have functional anti-IgE receptor antibodies and/or anti-IgE antibodies that can degranulate skin mast cells or basophils. In acute attack, about 58% patients have autoantibodies of IgE molecules, and 25% patients have anti-IgE antibodies. The increase of IgE antibodies in serum is the main reason for the secretion of inflammatory mediators such as histamine. Some cases have reported that anti-IgE antibodies and anti-leukotriene preparations are effective in treating some chronic urticaria.
Treatment of chronic urticaria
The severity and clinical types of chronic urticaria are quite different, and the treatment scheme should be individualized. As with other allergic diseases, if there are definite allergens, the treatment plan should include avoiding contact with allergens.
Antihistamines are commonly used in clinical treatment of urticaria. 1 generation Ruchlorpheniramine can effectively relieve symptoms and reduce the number of skin lesions, but it has adverse reactions such as drowsiness and anticholinergic. The second generation, such as loratadine and cetirizine hydrochloride, have weak sedative effect and are currently the first-line clinical drugs. However, we will find that antihistamines have a quick effect on urticaria, but they can be controlled after taking the medicine, and urticaria will still occur without taking the medicine. From acute urticaria to chronic urticaria, drug treatment is intermittent, and it still can't get rid of urticaria. In recent years, through the research on allergic diseases and IgE-mediated allergies, anti-IgE antibody immunotherapy has been introduced into some chronic allergic diseases, such as chronic urticaria, atopic dermatitis (eczema), perennial rhinitis and cough variant asthma.
Kangminyuan anti-allergic probiotics can participate in allergen-specific IgE-mediated immune allergic reaction. After human dendritic cells were co-cultured with anti-allergic probiotics, the immune function of Th 1 secreted by interleukin (IL- 12) was regulated, so as to screen anti-allergic probiotic strains with anti-allergic ability. Kangminyuan antiallergic probiotics improve the ecological balance of intestinal microorganisms, the largest immune organ of human body, by using active and non-pathogenic microorganisms, thus promoting immune tolerance. Lactobacillus salivarius has the international patent certification for reducing serum IgE allergic antibody, and it is continuously supplemented ...&>
Question 6: Ask for help. I have urticaria for four months. How to treat it? Thank you, God help me. I feel itchy every time I sleep recently, and then I get pimples. I hear it's a scratch disease. It has never happened before. What is the cause of this disease? How to treat it? How can you alleviate the symptoms? Help! thank you