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1. Hyperuricemia and gout hyperuricemia are the pathogenesis of gout, but they are not enough to cause gout. Gout only occurs when urate is deposited in body tissues and causes damage; The higher the serum uric acid level, the greater the possibility of gout in the next five years. At the onset of acute gouty arthritis, the serum uric acid level is not always high.
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2. Hyperuricemia and hypertension At present, many epidemiological studies have confirmed that serum uric acid is an independent risk factor for hypertension. Serum uric acid level increased by 59.5μmol/L, and the relative risk of hypertension increased by 25%. Clinical studies have found that 90% of patients with primary hypertension are complicated with hyperuricemia, while only 30% of patients with secondary hypertension are complicated with hyperuricemia, suggesting that hyperuricemia has a causal relationship with primary hypertension.
3. Hyperuricemia and diabetes Long-term hyperuricemia can destroy the function of pancreatic β cells and induce diabetes, and studies have confirmed that long-term hyperuricemia has a causal relationship with abnormal glucose tolerance and diabetes.
4. Epidemiological data of hyperuricemia and hypertriglyceridemia at home and abroad show that there is a correlation between serum uric acid and triglyceride. A prospective cohort study on the relationship between uric acid and triglyceride found that basal triglyceride is an independent predictor of hyperuricemia in the future.
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5. Hyperuricemia and metabolic syndrome The pathophysiological basis of metabolic syndrome is hyperinsulinemia and insulin resistance. Insulin resistance increases blood uric acid production during glycolysis and free fatty acid metabolism, and directly leads to hyperuricemia by increasing renal reabsorption of uric acid. 70% patients with metabolic syndrome have hyperuricemia.
6. Hyperuricemia and uric acid in coronary heart disease are independent risk factors for coronary heart disease death: Studies have shown that uric acid is an independent risk factor for coronary heart disease death in the general population, regardless of gender. With the increase of serum uric acid 1mg/dl, the death risk of men increased by 48%, and that of women increased by 1.26%. Serum uric acid > 357 μ mol/L is an independent risk factor for coronary heart disease. Serum uric acid > 4 16.5 μ mol/L is an independent risk factor for stroke.
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7. Hyperuricemia and renal damage Uric acid is closely related to renal diseases. In addition to uric acid crystal deposition leading to renal arterioles and chronic interstitial inflammation, many epidemiological investigations and animal studies have shown that uric acid can directly cause microvascular lesions of glomerular afferent arterioles, leading to chronic kidney diseases.
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